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The Imprinted Brain Hypothesis

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The Imprinted Brain Hypothesis

The imprinted brain hypothesis is based on a conflict of theory between the paternal and maternal genes set. This is explained during the gestation period in the division of labor between parents. Mothers tend to contribute almost everything from the gestation stage to birth and the lactation process. On the other hand, the fathers are only involved in the nuclear DNA composition, which therefore creates a genetic conflict in parental investments. A conflicting genetic interest is therefore developed whereby the mother is more interested in minimizing their investment, whereas the father’s interest is on maximizing it (Badcock, 2011). However, the mother’s genes could be existent in all her offsprings save for one that could inherit their fathers’ genes. Badcock & Crespi (2008) state that, in line with the imprinted brain theory, the paternal genes lead to the development of a child with a larger brain that is accustomed to making demands. When this becomes extreme, it is diagnosed as autism.

Autism and Schizophrenia are termed to be neurodevelopment genetic conditions that are hereditary and passed on to off sprigs through their genetic factors. Studies reveal that males are more prone to these disorders as compared to women due to their sex differences in a ratio of 4:1. The two disorders are acquired through genetic imprinting, whereby they develop through an expression of a gene and not inheritance (Badcock, 2011). Different theories have been raised in relation to the sex biases in these disorders, which some have been out ruled through human studies. Badcock & Crespi (2008) further state that depending on the genes programmed for expression, these conditions may be incurred, and they tend to differ on their sexism. Scientists have not been able to fully arrive at the etiology that results in autism, but it is noted that the condition is in favor of the paternal bias. Autism is associated with having a larger brain size, whereas Schizophrenia to slow brain development. The imbalances and maternal biases in the chromosome development through gene expression. Therefore, high paternal biases lead to hyper-mental deficits, thus leading to autism and Schizophrenia disorders.Autism and Schizophrenia are termed to be neurodevelopment genetic conditions that are hereditary and passed on to off sprigs through their genetic factors. Studies reveal that males are more prone to these disorders as compared to women due to their sex differences in a ratio of 4:1. The two disorders are acquired through genetic imprinting, whereby they develop through an expression of a gene and not inheritance (Badcock, 2011). Different theories have been raised in relation to the sex biases in these disorders, which some have been out ruled through human studies. Badcock & Crespi (2008) further state that depending on the genes programmed for expression, these conditions may be incurred, and they tend to differ on their sexism. Scientists have not been able to fully arrive at the etiology that results in autism, but it is noted that the condition is in favor of the paternal bias. Autism is associated with having a larger brain size, whereas Schizophrenia to slow brain development. The imbalances and maternal biases in the chromosome development through gene expression. Therefore, high paternal biases lead to hyper-mental deficits, thus leading to autism and Schizophrenia disorders.

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